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The two approaches may be complementary rather than mutually exclusive

It is that theres evidence transposition: animal data becomes human claims, a cousin molecule becomes this molecule, a mechanism becomes a protocol, one indication becomes a totally different use

But the math is simpler than it looks

Validation of transient elastography and comparison with spleen length measurement for staging of fibrosis and clinical prognosis in primary sclerosing cholangitis

It's noteworthy that these Parkinson-like events following ranibizumab administration are distinguished by the absence of cortical and subcortical atrophy, suggesting a unique pathophysiological mechanism distinct from typical PD presentations (Sultana and Scondotto 2020)