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Another detailed aspect of combination therapy involves the use of TGF- inhibitors alongside agents targeting other key signaling pathways implicated in fibrosis, such as the Wnt/-catenin or PI3K/Akt pathways (128)

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Through GPIb and GPIIb/IIIa, platelets bind to the staphylococcal accessory regulator protein (SarA) and Iron-responsive surface determinant B (IsdB), respectively ( S
