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Further, excessive Hcy can be converted to a Hcy thiolactone that can react with the -amino group of a protein lysine residue (N - homocysteinylation) and contribute to the manifestations of HHcy [31]
It acts as a primary aqueous-phase antioxidant, scavenging free radicals in the blood and extracellular fluid
Recent studies have shown that several mutations in RNA-binding proteins (RBPs) can also cause ALS
doi: 10.3389/fpls.2022.1040532 166 SlotM.GarciaM
