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As ferroptosis moves toward therapeutic reality, CoQ 10 oxidoreductases, particularly FSP1, stand out as a promising class of targets that may enable tumor-selective weakening of antioxidant defenses to unlock ferroptosis-based cancer treatment

TaoLWangJWangKLiuQLiHXuSet alExerkine FNDC5/irisin-enriched exosomes promote proliferation and inhibit ferroptosis of osteoblasts through interaction with Caveolin-1

Free Hcy, as well as amino acids, can enter endothelial cells through exchange transport systems, which increases its intracellular level

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The deposition of misfolded proteins within the ER lumen sets off a series of cascades aimed at restoring intracellular homeostasis (Fig