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The neuroprotective mechanisms of MEM in AD against neuronal loss (apoptosis decrease and mitochondrial alteration) and reduction of A production could be attributed, at least in part, to its selective action blocking extrasynaptic NMDAR

The novel point was the rapid activation of the collateral rescuing pathways (i.e., activation of the azygos veins direct blood flow delivery) to reestablish the reorganized blood flow [4,10]

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