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Description
40-45 The proposed mechanism of action is intravascular sequestration of the toxic drug to a newly formed intravascular lipid phase (lipid sink), decreasing the free blood concentration of the lipophilic drug
Current Biology, 12 , 427433
[132] [142] It has been implicated in addictions to alcohol, cannabinoids, cocaine, methylphenidate, nicotine, opioids, phencyclidine, propofol, and substituted amphetamines, among others
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