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Further evidence supporting the potential in situ deleterious effects of APAP in brain comes from studies showing that APAP increases neuronal CYP2E1 enzymatic activity and protein levels, leading to neuronal cell death through mitochondrial-mediated mechanisms that involve cytochrome c release and caspase 3 activation (130)

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Thiamine deficiency can lead to more downstream deficiencies by lowering our ability to absorb vitamins and minerals from the foods we eat.120 Cases of protein, fat or carbohydrate malabsorption can be caused by thiamine deficiency

Footnotes Disclosure The authors report no conflicts of interest in this work

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