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While one upregulates growth hormone receptors and accelerates angiogenesis at the immediate site of damage, the other acts systemically to mediate actin polymerization

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[ 164 ] Thus, BACH1 may act as a central hub, bringing together NANOG, SOX2, and OCT4 to stabilize these factors, and meanwhile, it increases the transcription of CD44 and CD133, ultimately contributing to the enhancement of cancer stem celllike properties

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