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Second, the effects appear to be centrally mediated rather than just a consequence of feeling sick or full

In addition to ADH release, hypovolemia-induced renal hypoperfusion stimulates the juxtaglomerular apparatus to secrete renin, activating the renin-angiotensin-aldosterone system

During hepatic fibrosis, however, HSCs become the major extracellular matrix producing cell type, with a predominant production of collagen type I (Maher and McGuire, 1990)

In support of this mechanistic insight, it has long been established that induction of diabetes in rats with STZ or alloxan favors uncontrolled availability of lipid peroxidation products, while consequently suppressing the intracellular antioxidant defences (Maritim et al., 2003