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Yang and colleagues investigated the role of the autophagy protein p62/SQSTM1 in regulating ROS generation during ferroptosis

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Therefore, further research is needed not only in astrocytes and neurons, but also in other glial cells in order to fully comprehend taurine metabolism and function in the brain

In ARDS, an imbalance between these pathways contributes to exaggerated immune responses, tissue damage, and disease progression ( 2.1 Ferroptosis: a central player in ARDS pathogenesis Ferroptosis is a regulated, iron-dependent form of cell death distinguished by oxidative damage and lipid peroxidation