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demonstrated that the GLP-1 analogue lixisenatide decreases atherosclerosis in insulin-resistant Apoe/Irs2+/ mice by promoting macrophage polarization towards an anti-inflammatory M2 phenotype, which was associated with enhanced plaque stability (i.e., thicker fibrous caps, fewer necrotic cores, and decreased inflammatory infiltrates)

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Diabetic wound healing studies using collagen dressings with GHK demonstrated a 9-fold increase in collagen synthesis with better epithelialization and activation of fibroblasts and mast cells