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[DOI] [PubMed] [Google Scholar] 74.Mulay SR, Kulkarni OP, Rupanagudi KV, et al

Increasing fatty acid and ketone metabolism after a ketogenic diet also attenuated reductions in ATP, NAD, mitochondrial fragmentation, and myelination in a murine diffuse axonal injury model, while treatment with ketone bodies reduced neuronal/myelin damage induced by glucose deprivation in vitro [96]

This explains why intracellular oxidation kinetics for roGFP2 fusion constructs with glutaredoxin mutants showed excellent correlation with the second order rate constants of the oxidative half-reaction from in vitro experiments 15,26,34,35

Moreover, UCH-L1 could be utilized to distinguish between individuals with PD and normal people, with the AUC of 0.89 (Mondello et al., 2014)