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Activating SIRT3 in adipocytes could be a potential strategy to alleviate obesity-related metabolic diseases.
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Levetiracetam, an antiepileptic agent, binds the synaptic vesicle protein SV2A
In this essay, we review the recent progress in the understanding of the mechanism underlying cuproptosis, revisit the roles of p53 in metabolic regulation and iron-sulfur cluster and glutathione biosynthesis, and propose several potential mechanisms for wild-type and mutant p53-mediated cuproptosis regulation.
