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To investigate whether S-nitrosylation of MYCN at specific cysteine residues contributes to NO-mediated suppression of ER stress in NEPC, we generated site-directed mutations at Cys4, Cys186, and Cys464 (Supplementary Fig

Its proposed mechanisms involve multiple cytoprotective pathways including upregulation of VEGF and growth factors promoting angiogenesis, modulation of the nitric oxide system, activation of the FAK-paxillin pathway for cell migration and tissue repair, interaction with the gut-brain axis affecting dopaminergic and serotonergic systems, and gastroprotective effects against NSAID and alcohol-induced mucosal damage

doi: 10.1016/j.cca.2007.01.026 254 ShafieiMSShettySSchererPERockeyDC

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And unlike men in the U.S., the Bolivian foragers-farmers do not show declines in testosterone with age