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In an effort to investigate whether NMN treatment would have an impact on the health of UMNs diseased with TDP-43 pathology, we utilized dissociated motor cortex cultures established from prpTDP-43 A315T -UeGFP mice, in which CSMN are genetically labeled with eGFP expression that is stable and long-lasting, and when in culture retain their CSMN identity and eGFP expression

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J Immunol 172:442448

demonstrated that blocking vascular endothelial growth factor (VEGF) signaling with antibodies against VEGF or VEGFR-2 reduced M.tb transmission to the lung, spleen, and liver in M.tb -infected mice [112]

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