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Medicines used in the treatment of hyperthyroidism have also been shown to have potential beneficial effects in relieving depressive symptoms

Expectedly, depletion of cystine or inhibition of the Xc- antiporter using synthetic small molecule inhibitors including erastin, sulfasalazine, sorafenib and even excess of glutamate triggered cystine depletion, decreased GSH level and reduced GPX4 activity, culminating in ferroptosis induction

The goal is consistent, effective dosing that produces predictable research outcomes

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These effects correlate with improved neurostructural outcomes, including restoration of motor coordination and behavioral performance in controlled laboratory settings