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Description
In murine studies, deletion of the IGF-1R in skeletal muscle resulted in glucose intolerance issues ultimately leading to T2D at an early age, because, although they express insulin receptors, they cannot form hybrid or IGF-1 receptors [162]
Furthermore, IGFBPs other than IGFBP-3 may play a role in the access of IGF-I to various tissues (129)
This can support faster absorption and results
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