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Ren Z-X, Brewton RG, Mayne R

Relevant to our interest, identical KRAS mutations were also found to exist in both adenomyotic and endometriotic lesions from several patients with co-occurring disease (Inoue et al., 2019)

We hypothesized that selenomethionine might directly participate in ROS scavenging

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We find that, although GSH cannot protect against pink-1 or pdr-1 loss-of-function in the context of -synuclein alone, the joint additive neurodegenerative phenotype of both pink-1 and pdr-1 was partially rescued by GSH (Figure 4f) to a degenerative state reminiscent of loss of pink-1 or pdr-1 alone