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Optimizing glutathione levels has been proposed as a strategy for health promotion and disease prevention, although clear, causal relationships between glutathione status and disease risk or treatment remain to be clarified

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The iron overload-induced oocyte damage and embryo toxicity might be related to ferroptosis mediated by multiple signaling pathways, including nuclear receptor coactivator 4-dependent ferritinophagy (123), glutathione peroxidase 4-dependent downregulation (124), and heme oxygenase 1 (HMOX1) overexpression (120), in which mitochondrial dysfunction and structural damage play a central role

This mechanism is supported by observations wherein inhibition of the electron transfer chain suppresses the ferroptosis caused by Cys deprivation [155,156]

Melanin can be found in the dermis