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Description
also called glycolate oxidase)
doi: 10.3389/fimmu.2023.1322746
Numbers displayed on the top left of each panel represent the animal identification number
Baseline pulmonary function tests were performed with standard medication withholds
These results suggest that perturbation of the malate/aspartate shuttle results in cytosolic retention of NADH and reductive stress, decreasing NADH availability in mitochondria and possibly increasing NAD + levels in the cytosol, thereby leading to inhibition of NADH-dependent mitochondrial respiration and enhancement of NAD + -dependent glycolysis
