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Current Neuropharmacology, 16(5), 566-583
These components act through a variety of mechanisms including decreasing inflammatory mediator production through effects on cell signalling and gene expression, reducing the production of damaging oxidants and promoting gut barrier function and anti-inflammatory responses (Figure 3)
As it turns out, an abundant exogenous source of a single nutrient can in some cases restore metabolic balance, normalize metabolic defects, and revert the antibiotic-resistant phenotype to antibiotic sensitivity 10,14,30
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