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In fact, busulfan, through interaction with glutathione and sulfhydryl groups and disruption of balance between ROS and antioxidant, increases oxidative stress and causes extensive intracellular damages to proteins, nucleic acids, and lipid molecules

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Rats treated with ALCAR (300 mg/kg) or propionylcarnitine (300 mg/kg) after ischemia had less loss of ATP and glutathione, more intact neurons, and decreased markers of oxidative stress and energy depletion in the hippocampal CA1 region 7 days after ischemia [133]