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Description
IGF-I signaling is mediated via its binding to the IGF-I receptor (IGF-IR), a transmembrane protein with tyrosine kinase activity[2] and activator of the PI3K/Akt pathway [4]
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158,159 The SARS-CoV-2 N protein disrupts the interactions between STAT1 and JAK1, as well as between STAT2 and TYK2, by competitively binding to both STAT1 and STAT2
doi:10.1111/j.1749-6632.2010.05853.x Xu J, Qiu JC, Ji X, et al
All three are recoverable
