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Description
APC, adenomatous polyposis coli
Alzheimer mechanisms and therapeutic strategies
Although it can alleviate systemic lipotoxicity and promote the clearance of apoptotic debris, this protective effect can also be offset by its potential to overactivate the SHP1/BTK axis, further leading to damage to FAO and exacerbating the transition to the immune paralysis stage
The pharmacological efficacy of baicalin in inflammatory diseases
The de-S-nitrosylation of G6PD could be a compensatory response to decreased NO and increased ROS generation during EC senescence
