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[DOI] [PMC free article] [PubMed] [Google Scholar] 86.Morotti M., Bridges E., Valli A., Choudhry H., Sheldon H., Wigfield S., Gray N., Zois C.E., Grimm F., Jones D., et al

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Whereas we have evidence for stimulation of mitochondrial ROS formation starting at 4d of noise exposure that is based primarily on mitoSOX fluorescence and an incomplete inhibition of cerebral ROS formation by genetic deletion of Nox2 (Kroller-Schon et al., 2018), a role of xanthine oxidase-dependent ROS formation can be assumed from the beneficial effects of the xanthine oxidase inhibitor allopurinol on cochlear damage and hearing loss in response to 60 h of loud noise exposure with a sound pressure level of 90 dB (Seidman et al., 1993) or 125 dB for 1.8 h (Cassandro et al., 2003)

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