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The brain compensates for stroke damage through neuroplasticity, forming new connections that bypass the damaged area

Sari, G

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Following this, a series of studies have explored its effects in various models of age-related diseases, focusing on atherosclerosis and neurodegeneration

For KEGG, the potential targets involved in ANH treatment were significantly associated with the HIF-1 signaling pathway, IL-17 signaling pathway, Th17 cell differentiation, and PI3K-AKT signaling pathway (Fig