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Mitochondrial respiratory capacity modulates LPS-induced inflammatory signatures in human blood

After pharmacological intervention in 96-well cell culture plates for 24, 48 or 72 h, the culture medium was removed and replaced with 0.1 mg MTT reagent (Beyotime, ST1537) per well dissociated in 100 L basal DMEM (Procell, PM150210)

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Supplementation of mice with FLAVOmega (a mixture of flavonoids and docosahexaenoic acid) ameliorated the pathological phenotype in dystrophic mice, as reflected by decreasing ROS production, suppressed inflammation and fibrosis, an oxidative metabolic switch of myofibrosis as well as increased mitochondrial activity, fatigue resistance, and vascularization