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Cooper AJL (2013) Possible treatment of end-stage hyperammonemic encephalopathy by inhibition of glutamine synthetase

8, an ultra-shortlived oxidant that inflicts site-specific macromolecular damageDNA base modifications and strand breaks near metal centers, protein carbonylation and initiation of lipid peroxidationwith risk amplified by iron mobilization (e.g., ferritinophagy) and disruption of FeS clusters (Zhang et al