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PRMT5 is highly expressed in breast cancer and chronic myeloid leukemia (CML) stem cells, and the knockdown of PRMT5 or the use of PRMT5 inhibitors can significantly impair the self-renewal capacity of CSCs

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Personalized dosing respects the fact that one patient's physiology, tolerance, and risk profile may differ from another's

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Zn synergistically enhances this transition via HSP70-mediated stress signaling, while MeHg induces S100B-overexpressing reactive astrocytes that secrete matrix metalloproteinases (MMPs) to degrade BBB components (94, 95)