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Available data suggest the ability of PS-NPs particles to induce lipophagy in hepatocytes via the AMPK/ULK1 pathway and potentiate lipid accumulation in them
doi: 10.1016/j.ccr.2012.11.020
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When this phenotype was fully corrected by introducing either HA-ATG2A or HA-ATG2A-3CS, overexpression of ZDHHC11 counteracted the effects of HA-ATG2A but not HA-ATG2A-3CS (Fig
13 Colistin resistance According to recent data, a significant percentage of infections caused by CRE in the USA required the use of colistin and polymyxin B, which are considered last-resort drugs
