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This phenomenon, known as reperfusion injury, involves the oxidation of cellular components, mitochondrial dysfunction, and the activation of inflammatory pathways [91, 92]

Chowdhury A, Nabila J, Adelusi Temitope I, Wang S (2020) Current etiological comprehension and therapeutic targets of acetaminophen-induced hepatotoxicity

G., Argyros, D
doi: 10.1002/lary.22157

Therefore, JAB1 associates with the CRL4B complex to jointly transcriptionally repress these target genes
