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In the pre-treatment studies, NaBPC157 delayed and prevented the arrhythmia occurrence and reduced and/or prevented ischemia

MOTS-c also regulates nuclear gene expression beyond AMPK, affecting glucose metabolism and oxidative capacity at the transcriptional level

The first argument that disputes the progenitor function is the considerable difference in cell morphology and size of CLCs (1723 m) and NCs (2585 m) [1, 8]

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