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The result is a 16-amino acid peptide that stimulates lipolysis through beta-3 adrenergic receptors on fat cells, without triggering IGF-1 elevation, insulin resistance, or the growth-promoting effects that make full HGH problematic for long-term metabolic use

All differences were statistically significant (CohBar press release, Aug 2021)

Individual protocols must be personalized based on body composition, metabolic status, and physician assessment

This represents a fundamental safety distinction from full-length human growth hormone, which consistently elevates IGF-1 through traditional growth hormone receptor activation