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2022;814 doi: 10.1016/j.scitotenv.2021.152425

Consistent with our findings, previous studies have highlighted the cytoprotective role of HIF-1 in ischemia-reperfusion injury and acute kidney injury through the activation of receptor-mediated mitophagy pathways [45, 46]

We hypothesized that loading water soluble antioxidants into the hypotonic, gel-forming vehicle could provide therapeutic scavenging of ROS in the posterior segment, a potential therapeutic opportunity for treating RP

However, when glutamine metabolism is targeted with a single treatment, tumor cells will activate other signaling pathways or strengthen glutamine metabolism reprogramming to meet their energy demand, resulting in different degrees of drug resistance in tumor cells, thus restricting targeted glutamine metabolism therapy