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Other clinically relevant species, such as Escherichia coli , Salmonella enterica , Neisseria gonorrhoeae , and Mycobacterium tuberculosis , also pose serious therapeutic challenges and contribute significantly to the global AMR burden [6, 7]

They display differences in their mechanisms of action, likely mediated by their binding to different, non-overlapping epitopes of the CD38 molecule

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Bpc-157 enhances this cell migration process, essentially speeding up the recruitment of healing resources to damaged areas