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FIGURE 12 Collectively, these findings indicate that GO@GSH-FA effectively preserves neuronal integrity and reduces apoptosis in a cerebral Ischemia-Reperfusion injury model, demonstrating its potential as a therapeutic agent for neuroprotection

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Total glutathione measured in HEK-293T cells overexpressing vector, WT hCHAC1, E115A CHAC1 mutant, or E115Q CHAC1 mutant

Specific reasons why remain unknown, but it is likely that some of the counter apoptotic mechanisms that occur during the development of T-cell memory or the process of chronic HIV infection are at least partially responsible