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AKT, a signaling molecule of the protein kinase B family, is activated in many human cancers and is part of the phosphatidylinositol 3-kinase pathway
Decreased ATP levels and altered expression of the pro-apoptotic protein BH3-interacting domain death agonist (BID) suggested mitochondrial dysfunction, which, together with ferritinophagy, contributed to lung tissue damage
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Effects vary and depend heavily on proper dosing and supervision
Reduced energy production may contribute to neuronal dysfunction and synaptic loss
