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In silico analysis evidenced that SSTR2 was a putative target of miR-185 and, intriguingly, miR-185 was upregulated in tumor samples from SSA non-responder adenoma patients with respect to controls while it was downregulated in the SSA responder patients (48)

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Although most UGA-containing transcripts do not facilitate Sec incorporation, transfer of a 3 UTR with a SECIS element to heterologous gene transcripts with a UGA codon embedded in an open reading frame is sufficient to direct Sec incorporation (217, 219, 322)

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