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In case of high TG availability, liver secretes larger TG-rich VLDL1 and TG-poor VLDL2 particles, which are converted to LDLIV and LDLII, respectively (Figure 2) (Hirayama and Miida, 2012

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Furthermore, the apoC-II mimetic peptide potentiated LPL activity in other non-apoC-II deficient HTG conditions, suggesting a potential broader utility 93

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