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However, a similar hypersensitivity to H 2 O 2 was also reported for the mutants with deletions of subunits of mitochondrial CIII ( cor1 , cyt1 ), suggesting that the elevated O 2 and the sensitivity to H 2 O 2 are more likely to be attributable to a consequence of respiratory dysfunction rather than a lack of the antioxidant role of CoQ (330)

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