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Therefore, ACSL4 is a key promoter determining the content of oxidizable PUFAs in membrane phospholipids, thereby facilitating ferroptosis (Liu J
DB treatment induced an obvious inhibition in cell growth of the human normal liver epithelial cells, while this was rescued following the adding of APS (Fig
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HGH acts as a master hormone triggering growth throughout the body by stimulating the liver to produce insulin-like growth factor 1 (IGF-1)
