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The first 8 weeks were great

doi: 10.1016/j.bbagen.2013.02.019

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We next examined the role of vCD44BP in vivo by comparing viral loads after infection with wild-type MCMV, the vCD44BP mutant, and a revertant virus in which vCD44BP expression was restored (REV)

Therefore, inhibition of NF-B and downstream effector molecules is a possible underlying mechanism of the antitumor and anti-angiogenic activity of TQ in osteosarcoma[57]