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In addition, Xu et al

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doi: 10.1111/pcmr.13040 74

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Notably, pathogens have evolved sophisticated strategies to exploit TBK1 for immune evasion ( C

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hypoxia, nitric oxide, and carbon monoxide), and inhibition of DosRST decreases the reservoirs of persistent drug-resistant bacteria in the host (Zheng and Abramovitch, 2020)

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Thus, we propose that the GSHhematin adducts formation is a protective mechanism of RBCs from the auto-oxidation of hemoglobin to prolong their survival

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