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Oxidative stress synergizes with inflammatory processes in AT to severely disrupt glucose homeostasis
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and (3) studies focusing on bone diseases unrelated to metabolic or microbiota-driven pathways (e.g., traumatic fractures without a systemic context)
Moreover, high -tocopherol levels could suppress the tumor inhibitory effect of - and -tocopherols by competing for their binding to the proteins required for chemopreventive effect [97]
