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Peters J

GSH is known to modulate DNA synthesis by maintaining reduced glutaredoxin or thioredoxin, which are required for the activity of ribonucleotide reductase, the rate-limiting enzyme in DNA synthesis (Holmgren, 1981)

Namba, K., Osawa, A., Ishizaka, S., Kitamura, N

84 The initial hypothesis that inducible nitric oxide synthase (iNOS) in Kupffer cells is a significant source of NO formation 85 was quickly dismissed, as iNOS was not consistently induced in different models 86 and iNOS inhibitors did not provide protection

Mitochondrial apoptosis effectors DIABLO and HTRA2, the oxidative-stress-responsive tumour suppressor PML, and SOD2 were upregulated