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In recent years, ferroptosis has emerged as a promising target for countering highly aggressive malignancies, and is an iron-dependent form of regulated cell death associated with accumulation of reactive oxygen species (ROS) and increased lipid peroxidation 7
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In HG-induced or SOD2-silenced H9C2 cells, APS improved cell apoptosis and higher levels of ROS, and regulated the levels of oxidative stress injury indicators 8-OH-dG and nitrotyrosine (Sun et al., 2019)
