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Plasmid-encoded IL-15 was shown to be well-tolerated in humans but offered no apparent augmentation in a 2012 clinical trial for a HIV gag DNA vaccine ( Research on IL-15 has been limited in recent years due to its biological dependence on trans-presentation via IL-15R for optimal T cell activation (100103)

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illustrated that co-mutations of NF1 and TP53 can lead to MPNST in mouse models ( Gfap-Cre Nf1 flox/flox ) without developing the PN phase [42]

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