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[DOI] [PMC free article] [PubMed] [Google Scholar] 51.Miele L., Valenza V., La Torre G., Montalto M., Cammarota G., Ricci R., Masciana R., Forgione A., Gabrieli M.L., Perotti G., et al
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As mentioned above, c-Met is aberrantly activated by gene amplification, overexpression, mutation, binding to other ligands, autocrine signaling or abnormally high HGF levels in cancer
This peptide is frequently incorporated into models exploring how peptide-copper interactions support communication consistency across interconnected structural systems
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