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For instance, heme can cause surface epithelial cells to remain susceptible to cytotoxic damage that alters surface-to-crypt signaling, leading to hyperproliferation and ultimately hyperplasia of crypt cells in mice supplied heme (Brouwer et al., 2004)

J.2005Arsenic (+3 oxidation state) methyltransferase and the inorganic arsenic methylation phenotype.2042(April 2005),164169,0004-1008X 71

Role of NRF2 in protection against hyperoxic lung injury in mice

although responses vary and are generally less pronounced than with direct GH administration

These findings, along with the fact that the lung can normally maintain GSH for long periods even with the oxidant burden of CS, lend credence to the theory that disease symptoms could arise when adaptive responses fail or are dysfunctional