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The Jarisch-Herxheimer reaction: revisited

The plethora of structural information, together with the increasing power of computational facilities and refinement of binding prediction tools (110, 112, 119), open the possibility to design peptides/peptidomimetics able to target the translation complexes, with the aim to decrease protein synthesis of specific neuronal mRNAs and rescuing a healthy phenotype in individuals affected by FXS

33 , e2006057 (2021)

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Kremer et al., 2024) with an indispensable role of STAT3 activation due to methylation at a CpG site in the GFAP promoter (Takizawa et al., 2001), and in TET1/2/3-mediated DNA demethylation for establishing neural stem cell identity and enabling glial (e.g., astrocyte and oligodendrocyte) differentiation (MacArthur et al., 2024)